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During cellular stress, p53 activates and regulates cell cycle arrest and cellular senescence or apoptosis pathways, and plays roles in tumor suppression (63)

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Indeed, short-term (4 h) excess light at 1500 mol m 2 s 1 led to the accumulation of H 2 O 2 in the cytosol and along the tonoplast, but was absent in cell compartments after 2 weeks of excess light treatment (Heyneke et al