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Despite the significant overlap in lipid profiles and gene expression changes among the three self-sterile mutants, and the shared phenotype of resistance to Pseudomonas aeruginosa (PA14) infection, the three mutants exhibited dramatically different lifespans under normal culturing conditions

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doi: 10.1001/jamainternmed.2023.7268

They also demonstrated that dietary supplementation of apoE / mice with choline, TMAO or betaine drives atherosclerosis by upregulating the scavenger receptors CD36 and SR-A1 on macrophages, leading to increased cholesterol accumulation and formation of foam cells (Koeth et al., 2013)