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Calcitriol has been shown to enhance autophagic activity in podocytes and tubular cells by restoring LC3-II expression and autophagic flux, while concurrently reducing apoptosis through the modulation of Bcl-2 and Bax expression (21)

It supports Th1/Th2 balance, enhances T cell function and is anti-inflammatory

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It helps break down stored fat, increase energy expenditure, and enhance overall metabolic efficiency without affecting blood sugar levels

The drug's primary action involves binding to both GIP and GLP-1 receptors, which are naturally occurring incretin hormones that regulate glucose metabolism and appetite