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Crucially, interactions between NSW and specific polymorphisms in CRY2 and RORA further elevated BC risk, underscoring the complex interplay of genetic susceptibility and environmental circadian disruption in BC etiology 195

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Moreover, we assessed CoQ10 content and NAD + /NADH ratios, which are closely associated with FSP1s anti-ferroptotic function, PPARa over-expression not only increased FSP1 levels but also enhanced CoQ10 activity and elevated NADH content (Fig

Hauser Veronique Demers-Mathieu Brian Scottoline Manoj K