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The mechanism involves upregulation of growth hormone receptors in tendon fibroblasts and increased collagen synthesis at the repair site

All for one but not one for all: excitatory synaptic scaling and intrinsic excitability are coregulated by CaMKIV, whereas inhibitory synaptic scaling is under independent control

This indicates that GHRP-6 works both directly on the pituitary and indirectly through hypothalamic GHRH neurons, and the two pathways function synergistically

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Moreover, combinational drugs require synchronised delivery and consistent exposure are without compromising formulation integrity