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Therefore, while systemic fasting alone might not uniformly starve a tumor of monosaccharides, it could create localized nutrient shortfalls in parts of the tumor, potentially slowing progression in those microregions even if the overall tumor maintains sufficient glucose supply

have demonstrated that mutations in the precore region of the hepatitis B virus (HBV) genome affected the coding of HBV e antigen (HBeAg), which was associated with severe immunological dysregulation in acute severe hepatitis[34, 35]

Zhou L, Xue X, Hou Q, Dai C

BNIP3, BCL2 and adenovirus E1B 19-kDa-interacting protein 3